当血管壁受损，胶原蛋白和组织因子暴露，循环血小板通过血管性血友病因子的作用发生黏着，并伴形状改变和ADP的释放。
When vessel walls are damaged, collagen and tissue factor are exposed, and circulating platelets adhere via von Willebrand factor and undergo a change in shape with the accompanying release of ADP.
微栓塞能够引起组织缺血，红细胞片断引发溶血性贫血，或者血小板和凝血因子消耗导致出血。
Microscopic thrombi can produce tissue ischemia, red cell fragmentation leading to a hemolytic anemia, or hemorrhage due to consumption of platelets and clotting factors.
但是有一个可能是血液替代品里面的血红蛋白分子夺取氧化一氮分子，这些分子一般发挥舒张血管和组织血小板凝集作用。
But one possibility is that hemoglobin molecules in substitute blood capture nitric oxide molecules that normally dilate blood vessels and keep platelets from getting sticky.
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